The potential risk to human health of antibiotics that pass through the food chain has become an important global issue, but there are few reports on the response of ginger (Zingiber officinale) to antibiotic pollution. In this study, we investigated the enrichment characteristics and biological response of ginger to sulfamethoxazole (SMZ) and ofloxacin (OFL) residues, which are common in the environment. Lower levels of SMZ, OFL and their combined duplex treatment (SMZ+OFL) promoted the growth of ginger, but the critical doses necessary to stimulate growth differed among treatments 10&nbsp;mg&nbsp;L-1 SMZ, 1&nbsp;mg&nbsp;L-1 OFL and 1&nbsp;mg&nbsp;L-1 (SMZ+OFL) had the strongest stimulating effects. At higher dosages, the root growth and light energy utilization efficiency of ginger were impaired, and (SMZ+OFL) had the strongest inhibitory effect. Treatments with lower levels of antibiotics had no significant effect on reactive oxygen species and antioxidant enzyme activities. However, when SMZ, OFL and SMZ+OFL concentrations exceeded 10&nbsp;mg&nbsp;L-1, the contents of H2O2, O2- and MDA continued to increase, while the activities of SOD, POD, CAT first increased and then decreased, especially in SMZ+OFL. Ginger accumulated more SMZ and OFL in rhizomes and less in leaves, and accumulation increased significantly as antibiotic concentration increased. When SMZ concentration was 1&nbsp;mg&nbsp;L-1, the SMZ concentrations in rhizomes, roots, and leaves were 0.23, 0.15, and 0.05&nbsp;mg&nbsp;kg-1, respectively, and the residual SMZ in the rhizome was 2.3 times higher than the maximum residue limit. The abundance of the resistance genes sul1, sul2, qnrS, and intI1 increased with increasing antibiotic concentrations, and intI1 abundance was the highest. OFL induced higher levels of intI1 expression than did SMZ. Cadmium is toxic to plants, easily reaching unsafe levels for animal and human consumption. A greenhouse experiment investigated the effect of foliar-applied Cd on the accumulation and distribution of Cd, Zn, Cu and Pb in wheat (Triticum aestivum) grown in heavy metal-contaminated soil. Cadmium solutions (0, 10, 20, 30 and 40&nbsp;mg&nbsp;L-1) were repeatedly sprayed on entire aboveground wheat plants during heading stage to medium milk development stage. Plant sample analyses after harvest indicated that both the biomass yield and grain yield were negatively affected by Cd application (p&nbsp;&nbsp;0.05). Stem, leaf and glume Zn concentrations increased by less than 31%, while grain Zn concentrations were negatively affected by Cd treatments (3.4-34.4% lower than the control). Grain Cu concentrations were also negatively affected by Cd treatments, while grain Pb concentrations were similar between treatments. The antagonistic effect of Cd on grain Zn accumulation may mainly be due to competition for transporters and binding compounds in wheat leaves and stems. Preventing excess Cd from entering aboveground plant tissues should lessen negative plant and potentially animal/human health effects. In this study, 45 indoor dust samples and four particulate samples from air-conditioner filters were collected from four different indoor environments in Guangzhou, China, and the concentration and composition of organophosphate tri-esters (OPEs) and organophosphate di-esters (Di-OPs) were determined. Eight of the 10 target OPEs were detected in indoor dust at different detection frequencies (DFs), with tris(2-chloroisopropyl) phosphate and tris(2-chloroethyl) phosphate being the main components. Seven target Di-OPs were detected at different DFs, with diphenyl phosphate being the dominant compound. The total OPEs (?8 OPEs) and total Di-OPs (?7 Di-OPs) concentrations varied from 726 to 39,312&nbsp;ng/g and 68.8-14,766&nbsp;ng/g, respectively. The ?8 OPEs concentrations in instrumental houses were significantly higher (p&nbsp; less then &nbsp;0.001) than in three other indoor environments. The varying strengths of the correlation between Di-OPs and their respective parent OPEs was suggestive of their emission sources (e.g., direct application, impurities in OPE formulas, and OPE degradation). The hazard index (HI) values of individual OPEs in residential house were lower than 1, the results suggested a limited human health risk from individual OPEs. However, the total HI value (?HIs) of OPEs was approximately 1 based on a high exposure scenario and suggested a low risk for toddlers. Atmospheric wet deposition of base cations (BCs) plays a significant role in providing plant nutrients and buffering acidification. However, the temporal dynamic of wet BC deposition in China during the past two decades remain unclear. Here, we used long-term monitoring and literature data since 2000 to assess the temporal dynamics (seasonal and inter-annual variation), spatial patterns, main influencing factors, source apportionment, and capacity to neutralize the acidity of wet BC depositions at site, regional, and national scales. The results showed that total wet deposition of BCs was, on average, 2.12 keq ha-1 yr-1, where Ca2+ accounted for 65.57% of the total deposition, followed by Na+ (13.21%), Mg2+ (13.68%), and K+ (7.55%). Qinghai-Tibet had significantly lower BC deposition fluxes than northern, southern, and central China, as well as Inner Mongolia. Exchangeable BCs in soil, PM10 in the atmosphere, energy consumption, and cement production are significantly related to wet BC deposition, which accoucidification models. Polybrominated diphenyl ethers (PBDEs) are a series of highly persistent organic pollutants (POPs) ubiquitously distributed in marine environments. As key primary producers, microalgae are the start of PBDEs bioaccumulations and vulnerable to their toxicities. https://www.selleckchem.com/products/Sodium-butyrate.html In order to deeply investigate the toxic mechanism of PBDEs on microalgal cells, the occurrence of programmed cell death (PCD) in a model diatom Thalassiosira pseudonana and its possible mediating mechanism were studied. The results indicated cell death of T.&nbsp;pseudonana happened under the stress of BDE-47, which was proved to be PCD based on the correlations with three biochemical markers (DNA fragmentation, phosphatidylserine externalization and caspase activity) and three molecular markers [Metacaspase 2 gene (TpMC2), Death-associated protein gene (DAP3) and Death-specific protein 1 gene (TpDSP1)]; Furthermore, the changes of cellular ROS levels were correlated with the PCD markers and the dead cell rates, and the cell membrane and the chloroplast were identified as the major ROS production sites.